参考文献/References:
[1] JAGER RD,MIELER WF,MILLER JW.Age-related macular degeneration[J].N Engl J Med,2008,358(24):2606-2617.
[2] CONGDON NG,FRIEDMAN DS,LIETMAN T.Important causes of visual impairment in the world today[J].JAMA,2003,290(15):2057-2060.
[3] VINGERLING JR,DIELEMANS I,HOFMAN A,GROBBEE DE,HIJMERING M,KRAMER CF,et al.The prevalence of age-related maculopathy in the Rotterdam Study[J].Ophthalmology,1995,102(2):205-210.
[4] VELEZ-MONTOYA R,OLIVER SC,OLSCON JL,FINE SL,MANDAVA N,QUIROZ-MERCADO H.Current knowledge and trends in age-related macular degeneration:today’s and future treatments[J].Retina,2013,33(8):1487-1502.
[5] CATT RESEARCH GROUP.Ranibizumab and bevacizumab for neovascular age-related macular degeneration[J].N Engl J Med,2011,364(19):1897-1908.
[6] YOUNG M,CHUI L,FALLAH N,OR C,MERKUR AB,KIRKER AW,et al.Exacerbation of choroidal and retinal pigment epithelial atrophy after anti-vascular endothelial growth factor treatment in neovascular age-related macular degeneration[J].Retina,2014,34(7):1308-1315.
[7] LOIS N,MCBAIN V,ABDELKADER E,SCOTT NW,KUMARI R.Retinal pigment epithelial atrophy in patients with exudative age-related macular degeneration undergoing anti-vascular endothelial growth factor therapy[J].Retina,2013,33(1):13-22.
[8] GRUNWALD JE,DANIEL E,HUANG J,YING GS,MAGUIRE MG,TOTH CA,et al.Risk of geographic atrophy in the comparison of age-related macular degeneration treatments trials[J].Ophthalmology,2014,121(1):150-161.
[9] ROSENFELD PJ,BROWN DM,HEIER JS,BOYER DS,KAISER PK,CHUNG CY,et al.Ranibizumab for neovascular age-related macular degeneration[J].N Engl J Med,2006,355(14):1419-1431.
[10] MARTIN DF,MAGUIRE MG,FINE SL,YING GS,JAFFE GJ,GRUNWALD JE,et al.Ranibizumab and bevacizumab for treatment of neovascular age-related macular degeneration:two-year results[J].Ophthalmology,2012,119(7):1388-1398.
[11] ROFAGHA S,BHISITKUL RB,BOYER DS,SADDA SR,ZHANG K;SEVEN-UP STUDY GROUP.Seven-year outcomes in ranibizumab-treated patients in ANCHOR,MARINA,and HORIZON:a multicenter cohort study (SEVEN-UP)[J].Ophthalmology,2013,120(11):2292-2299.
[12] PEDEN MC,SUNER IJ,HAMMER ME,GRIZZARD WS.Long-term outcomes in eyes receiving fixed-interval dosing of anti-vascular endothelial growth factor agents for wet age-related macular degeneration[J].Ophthalmology,2015,122(4):803-808.
[13] BHISITKUL RB,MENDES TS,ROFAGHA S,ENANORIA W,BOYER DS,SADDA SR,et al.Macular atrophy progression and 7-year vision outcomes in subjects from the ANCHOR,MARINA,and HORIZON studies:the SEVEN-UP study[J].Am J Ophthalmol,2015,159(5):915-924.
[14] KREGEL KC,ZHANG HJ.An integrated view of oxidative stress in aging:basic mechanisms,functional effects,and pathological considerations[J].Am J Physiol Regul Integr Comp Physiol,2007,292(1):R18-36.
[15] TRACHOOTHAM D,LU W,OGASAWARA MA,VALLE NRD,HUANG P.Redox regulation of cell survival[J].Antioxid Redox Signal,2008,10(8):1343-1374.
[16] MALIK D,TAREK M,Del CARPIO JC,RAMIREZ C,BOYER D,KENNEY MC,et al.Safety profiles of anti-VEGF drugs:bevacizumab,ranibizumab,aflibercept and ziv-aflibercept on human retinal pigment epithelium cells in culture[J].Br J Ophthalmol,2014,98(Suppl 1):11-16.
[17] BEDARD K,KRAUSE KH.The NOX family of ROS-generating NADPH oxidases:physiology and pathophysiology[J].Physiol Rev,2007,87(1):245-313.
[18] QIU Y,TAO L,LEI C,WANDG J,YANG P,LI Q,et al.Downregulating p22phox ameliorates inflammatory response in Angiotensin Ⅱ-induced oxidative stress by regulating MAPK and NF-κB pathways in ARPE-19 cells[J].Sci Rep,2015,5(2):143-162.
[19] ZHOU W,QUAN JH,LEE YH,SHIN DW,CHA GH.Toxoplasma gondii proliferation require down-regulation of host Nox4 expression via activation of PI3 kinase/Akt signaling pathway[J].PLoS One,2013,8(6):e66306.
[20] CRESTANI B,BESNARD V,BOCZKOWSKI J.Signalling pathways from NADPH oxidase-4 to idiopathic pulmonary fibrosis[J].Int J Biochem Cell Biol,2011,43(8):1086-1089.
[21] LIU RM,DESAI LP.Reciprocal regulation of TGF-β and reactive oxygen species:a perverse cycle for fibrosis[J].Redox Biol,2015,6:565-577.
[22] BAUER KM,WATTS TN,BUECHLER S,HUMMON AB.Proteomic and functional investigation of the colon cancer relapse-associated genes NOX4 and ITGA3[J].J Proteome Res,2014,13(11):4910-4918.
[23] 伊文霞,韩颖,赵阳,张静,秦炯.内源性一氧化碳对反复热性惊厥脑损伤大鼠内质网应激PERK信号通路的调控作用[J].中华实用儿科临床杂志,2013,28(19):1502-1505.
YI WX,HAN Y,ZHAO Y,ZHANG J,QIN J.Regulating effect of endogenous carbon monoxide on brain damage induced by recurrent febrile seizures through GRP78-PERK-eIF2α-CHOP signal pathway[J].Chin J Appl Clin Pediatr,2013,28(19):1502-1505.
相似文献/References:
[1]范姜砾 王雨生 张鹏.湿性年龄相关性黄斑变性患者血浆中相关抗氧化酶水平测定[J].眼科新进展,2012,32(5):000.
[2]麻南 李丹 高付林 胡莲娜.玻璃体内注射贝伐单抗和贝伐单抗/曲安奈德联合用药治疗糖尿病性黄斑水肿疗效及安全性差异的荟萃分析[J].眼科新进展,2012,32(6):000.
[3]王毅 李罗翔 李娟 曾庆华.ApoE基因缺失小鼠视网膜及Bruch膜组织形态观察[J].眼科新进展,2013,33(1):000.
[4]徐新荣 仲路 黄冰林 魏源华 周欣 王玲 王富强.年龄相关性黄斑变性血浆蛋白质组学初步研究[J].眼科新进展,2013,33(2):000.
[5]党亚龙 陈彬川 穆雅林 赵满丽 朱豫.i-MP对产生2型脉络膜新生血管的年龄相关性黄斑变性患者视力及黄斑厚度的影响[J].眼科新进展,2013,33(2):000.
[6]王毅 李罗翔 李进辉 李娟 曾庆华.血脂异常ApoE基因缺失小鼠视网膜色素上皮细胞胞浆内黑色素和脂褐素的改变[J].眼科新进展,2013,33(7):000.
[7]罗文婷 孙大卫.血管黏附蛋白-1在眼科疾病中的研究进展[J].眼科新进展,2013,33(8):000.
[8]陆秉文 吴星伟.光动力疗法治疗年龄相关性黄斑变性的研究进展[J].眼科新进展,2013,33(4):000.
[9]栾兰 姚勇.湿性年龄相关性黄斑变性的治疗进展[J].眼科新进展,2013,33(4):000.
[10]胡艳红 祁明信 郭娜 陈胜 柯发杰.渗出型年龄相关性黄斑变性患者外周血CCR3的表达[J].眼科新进展,2013,33(11):000.
[11]张盼盼,董琪,华英彬,等.贝伐单抗对人视网膜色素上皮细胞形态、凋亡率及凋亡相关因子表达的影响[J].眼科新进展,2018,38(4):314.[doi:10.13389/j.cnki.rao.2018.0074]
ZHANG Pan-Pan,DONG Qi,HUA Ying-Bin,et al.Effects of bevacizumab on morphology and apoptosis of human retinal pigment epithelial cells as well as the expression of apoptosis-related factors[J].Recent Advances in Ophthalmology,2018,38(4):314.[doi:10.13389/j.cnki.rao.2018.0074]