参考文献/References:
[1] QUIGLEY H A,BROMAN A T.The number of people with glaucoma worldwide in 2010 and 2020[J].Br J Ophthalmol,2006,90(3):262-267.
[2] THORLEIFSSON G,WALTERS G B,HEWITT A W,MASSON G,HELGASON A,DEWAN A,et al.Common variants near CAV1 and CAV2 are associated with primary open-angle glaucoma[J].Nat Genet,2010,42(10):906-909.
[3] RONG S S,CHEN L J,LEUNG C K,MATSUSHITA K,JIA L,MIKI A,et al.Ethnic specific association of the CAV1/CAV2 locus with primary open-angle glaucoma[J].Sci Rep,2016,6:27837.
[4] KIM S,KIM K,HEO D W,KIM J S,PARK C K,KIM C S,et al.Expression-associated polymorphisms of CAV1-CAV2 affect intraocular pressure and high-tension glaucoma risk[J].Mol Vis,2015,21:548-554.
[5] LOOMIS S J,KANG J H,WEINREB R N,YASPAN B L,COOKE B J,GAASTERLAND D,et al.Association of CAV1/CAV2 genomic variants with primary open-angle glaucoma overall and by gender and pattern of visual field loss[J].Ophthalmology,2014,121(2):508-516.
[6] CHEN F,KLEIN A P,KLEIN B E,LEE K E,TRUITT B,KLEIN R,et al.Exome array analysis identifies CAV1/CAV2 as a susceptibility locus for intraocular pressure[J].Invest Ophthalmol Vis Sci,2014,56(1):544-551.
[7] GU X,FLIESLER S J,ZHAO Y Y,STALLCUP W B,COHEN A W,ELLIOTT M H.Loss of caveolin-1 causes blood-retinal barrier breakdown,venous enlargement,and mural cell alteration[J].Am J Pathol,2014,184(2):541-555.
[8] REAGAN A,GU X,HAUCK S M,ASH J D,CAO G,THOMPSON T C,et al.Retinal Caveolin-1 modulates neuroprotective signaling[J].Adv Exp Med Biol,2016,854:411-418.
[9] SETHNA S,CHAMAKKALA T,GU X,THOMPSON T C,CAO G,ELLIOTT M H,et al.Regulation of phagolysosomal digestion by caveolin-1 of the retinal pigment epithelium is essential for vision[J].J Biol Chem,2016,291(12):6494-6506.
[10] BERTA A I,KISS A L,LUKATS A,SZABO A,SZEL A.Distribution of caveolin isoforms in the lemur retina[J].J Vet Sci,2007,8(3):295-297.
[11] 孔蕾,胡敏,华启云,张红.实验性视网膜缺血-再灌注损伤中坏死性凋亡相关因子的表达变化[J].眼科新进展,2016,36(12):1109-1112.
KONG L,HU M,HUA Q Y,ZHANG H,Expression changes of necroptosis related factors in experimental retinal ischemia reperfusion injury[J].Rec Adv Ophthalmol,2016,36(12):1109-1112.
[12] NAG S,VENUGOPALAN R,STEWART D J.Increased caveolin-1 expression precedes decreased expression of occludin and claudin-5 during blood-brain barrier breakdown[J].Acta Neuropathol,2007,114(5):459-469.
[13] SHEN J,MA S,CHAN P,LEE W,FUNG P C,CHEUNG R T,et al.Nitric oxide down-regulates caveolin-1 expression in rat brains during focal cerebral ischemia and reperfusion injury[J].J Neurochem,2006,96(4):1078-1089.
[14] GAO Y,CHU M,HONG J,SHANG J,XU D.Hypoxia induces cardiac fibroblast proliferation and phenotypic switch:a role for caveolae and caveolin-1/PTEN mediated pathway[J].J Thorac Dis,2014,6(10):1458-1468.
[15] GAO L,CHEN X,PENG T,YANG D,WANG Q,LV Z,et al.Caveolin-1 protects against hepatic ischemia/reperfusion injury through ameliorating peroxynitrite-mediated cell death[J].Free Radic Biol Med,2016,95:209-215.
[16] ELLIOTT M H,ASHPOLE N E,GU X,HERRNBERGER L,MCCLELLAN M E,GRIFFITH G L,et al.Caveolin-1 modulates intraocular pressure:implications for caveolae mechanoprotection in glaucoma[J].Sci Rep,2016,6:37127.
[17] SURGUCHEVA I,SURGUCHOV A.Expression of caveolin in trabecular meshwork cells and its possible implication in pathogenesis of primary open angle glaucoma[J].Mol Vis,2011,17:2878-2888.
[18] CLARK R,NOSIE A,WALKER T,FARALLI J A,FILLA M S,BARRETT-WILT G,et al.Comparative genomic and proteomic analysis of cytoskeletal changes in dexamethasone-treated trabecular meshwork cells[J].Mol Cell Proteomics,2013,12(1):194-206.
[19] AGA M,BRADLEY J M,WANCHU R,YANG Y F,ACOTT T S,KELLER K E.Differential effects of caveolin-1 and -2 knockdown on aqueous outflow and altered extracellular matrix turnover in caveolin-silenced trabecular meshwork cells[J].Invest Ophthalmol Vis Sci,2014,55(9):5497-5509.
[20] LEI Y,SONG M,WU J,XING C,SUN X.eNOS Activity in CAV1 knockout mouse eyes[J].Invest Ophthalmol Vis Sci,2016,57(6):2805-2813.
[21] LI X,MCCLELLAN M E,TANITO M,GARTEISER P,TOWNER R,BISSIG D,et al.Loss of Caveolin-1 impairs retinal function due to disturbance of subretinal microenvironment[J].J Biol Chem,2012,287(20):16424-16434.
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